Who is the head of the household in Mosuo culture?
How is family lineage traced in Mosuo culture?
If an appropriate child is not born into a particular family, who then takes over the management of that family when the family leader dies or is no longer able to lead?
What is the primary religion of the Mosuo people?
For this situation contemplate we will consider three competitors who are externally comparable and have given wounds because of their game. The competitors will be alluded to as Mr.A, Mr. B and Mr. C. Each is in their twenties and are club standard sprinters. Mr.A fell amid a preparation run and supported reversal damage to one side lower leg. Mr. B gave a pre-patella bursitis of his correct knee and Mr. C couldn't contend in light of serious metatarsalgia. System and pathophysiology of damage On the off chance that we consider the etiology and systems of every damage we can see that in spite of the fact that they are to a great extent wears related and, to a degree sports particular, each is in a general sense diverse regarding introduction, cause, treatment and result. Give us a chance to think about Mr. A. who fell amid preparing. He was an unassuming club sprinter who ran sporadically for individual satisfaction. He supported intense reversal damage which brought about a halfway tear of the horizontal malleolar tendon. This tendon successfully joins the Tibia to the bone and calcareous and is to a great extent in charge of the horizontal soundness of the joint. (Clemente C D 1975). There was prompt agony and subcutaneous swelling and, in spite of the fact that he could weight bear instantly after the damage, Mr.A could just stroll with incredible torment. Parallel contortion of the joint was amazingly difficult. The central etiology of the damage was a sudden reversal worry to the lower leg which was more prominent than the tendon could withstand and this brought about burst of a portion of the collagen filaments of the parallel tendon together with the hidden joint case. This enabled significant seeping to track into the encompassing tissues which, together with both extravasation of synovial liquid and aggregation of tissue oedema, prompted the clinically obvious swelling over the sidelong malleolus. Mr. B, by differentiate, was a savagely focused sub-world class sprinter who saw his damage growing all the more gradually finished a time of around ten days. He was planning for a race and had expanded his running timetable both in power and separation secured. At first he knew about an uneasiness in the foremost part of his knee which felt shallow. This was clear toward the finish of his instructional meetings and held on for a couple of hours while voyaging home. As the instructional meetings heightened, the torment developed more relentless until the point when it happened all through his running session. Despite the fact that it was an annoyance, it was not extreme. Before ten days' over it had turned out to be extremely serious to the point that there was self evident swelling over the lower post of the patella which was delicate to the touch and always excruciating. The system of this damage is regular of the abuse damage seen with overtraining. It is accepted to emerge at first from miniaturized scale tears inside the body of the patella tendon which wind up aroused and the steady anxieties required with preparing don't give the damage a chance to mend adequately and the irritation ends up aggregate to the point that histology would indicate incendiary changes happening all through the tendon and this, thusly, causes contact on the encompassing structures. (Hewett T E et al. 1999) This is show as a steady dynamically agonizing swelling restricted in the locale of the patella tendon and is bothered by development of the knee joint. It is delicate to the touch and cutoff points work out. Mr. C was a club sprinter of unassuming capacity, yet with an over idealistic energy about his own particular capacity, who prepared with the tip top sprinters at the club. He oftentimes grumbled of minor wounds that were rebuked for his specific absence of execution in races. On this event he gave torments in his forefoot over the metatarsal heads which was unmistakable and happened when the toes were flexed yet not when they were broadened (an unphysiological finding). He could run, however griped intensely of forefoot torment after the race and could be seen tottering off the track and around the changing rooms after the race. Examination of his foot was totally unremarkable and no reliable physical irregularity could be found. It was recognizable that Mr. C vociferously rebuked this issue for his powerlessness to perform well. No physical finding was made yet the etiology of his dissension was believed to be a psychosomatic appearance of his uneasiness identifying with his failure to beat the better sprinters at the club. This likened to a component of psychological twisting and foreswearing together with a compensatory transformation side effect complex to justify his poor execution. (Patel D R et al. 2000) In short we see three aggressive sprinters with regular introductions of damage, yet three altogether different instruments of pathophysiology and etiology. Each will require an alternate way to deal with treatment and will take after an altogether different disease direction. Brain science of games damage There are various distinctive hypothetical ideas (with varying degrees of security of proof base) that can be conveniently utilized in depicting donning inspiration and are in this way significant to the occurrence of brandishing damage. (Wigfield An et al. 2000) The writing regarding these matters is exceptionally broad and past the extent of this exposition to consider in any level of detail. Inversion hypothesis (Apter M J 2001) is regularly used in such manner and can portray connections between the identity qualities and motivational boosts. Paratelic overwhelming competitors usually enter the paratelic motivational state and are normally excitement searchers and take part in high hazard and exceedingly aggressive games (viz Mr. B )(Cogan N An et al. 1998) Mr.A, by differentiate is the run of the mill telic predominant competitor who have a tendency to be excitement avoiders, who design and consider their preparation painstakingly and lean toward low force encounters. (Kerr J H et al. 1999) Give us a chance to begin this thought of the brain research of games damage with an evaluation of Mr. C who presents with a principally mental grievance Mr. C has an unmistakably psychosomatic introduction. This can be helpfully depicted as far as inversion hypothesis (Apter M J 2001). what's more, the paratelic idea (Murgatroyd S et al. 1978). There are parts of the metamotivational states depicted in the hypothesis which are pertinent to Mr. C's impression of his intentions in proceeded with investment in running notwithstanding when he was plainly neglecting to accomplish his set targets. On the off chance that gives the idea that Mr. C has built up a variety of a paratelic defensive system with substantial develops. He needs the high excitement delight of the paratelic overwhelming competitor by fixing up on the beginning line with the tip top competitors, yet has built up his particular phenomenological outline as a method for dealing with stress which permits him an impression of wellbeing from his view of disappointment with a progression of physical reasons for his inability to perform. (Kerr J H 2001) One mental system that has been shown to work in this kind of case is a type of psychological conduct treatment which permits an acknowledgment of the ramifications of an activity to be reexamined by the customer. (Fowler D et al. 1995) This was joined with a procedure of the setting of "well ordered" here and now objectives. This adequately enabled the customer to think about his need to unreasonably contrast himself and the world class competitors and to enable him to accomplish dynamic feasible targets, consequently perceiving and exploiting accomplishment instead of ruminating on poor past execution examinations with other (better) competitors. (Torment M et al. 2004). The thought is that by setting and accomplishing some fleeting objectives, the customer can center around the present, make little dynamic advances, and perceive new accomplishments, rather than ruminating on past execution level. (Lobby H K et al. 2001). Complicity by the clinician in concurring that his side effects may really be physical can be totally counterproductive in this sort of case (see on) Damage administration The protest of administration of any damage is obviously to amplify the level of recuperation conceivable and to utmost and leftover incapacity that may happen because of the damage. In expansive terms we can consider the prompt (emergency treatment) treatment and the consequent longer term administration as partitioned issues. (Hergenroeder A C 2003) On account of Mr.A's intense damage the basic components of treatment (once the analysis has been certainly made) ought to be to anticipate additionally tissue harm and seeping by immobilization of the joint (bracing), incite cooling to lessen the tissue response to the damage, absense of pain to mitigate the agony (yet with the proviso that relief from discomfort ought not be a sign to pressure the joint) and strain to limit blood and tissue liquid collection. The more drawn out term contemplations ought to be that weight bearing ought to be kept to a base for around 7-10 days. Assembly should then start in a reviewed form over around four to a month and a half. Running on level surfaces could reasonably start (conceivably with a lower leg bolster) after that time. Preparation (both dynamic and detached) is important to guarantee that the fibroblastic action of the tendon repair component does not limit development of the joint to the extent that the long haul limitation of development turns into an issue. (Plantation J 2003) Mr.A would be all around encouraged to abstain from running on uneven surfaces for a time of numerous months and to attempt a course of physiotherapy including modalities, for example, wobble load up preparing to enhance his proprioceptive capacities. (Lephart S M et al. 1997) Because of the damage, Mr.A ought to dependably see himself as more inclined to get a repeat if he somehow happened to have another fall. Mr. B ought to be dealt with in a particularly unique manner. There is no "intense" medical aid treatment all things considered, as the basic factor here is to perceive that the damage is the aftereffect of abuse of a joint. Rest, or at times basically a lessening in the preparation plan, is regularly all that is expected to permit the condit>GET ANSWER