The client is a 23-year-old African American male with type-1 diabetes who has not been taking his insulin as prescribed or adhering to a diabetic diet. The client presented to the emergency department with complaints of lethargy, abdominal pain, nausea, and malaise.
Vital signs:
• Temperature: 98.6 F
• Pulse: 110 beats/minute
• Respirations: 32 breaths/minute
• Blood pressure: 90/60 mm Hg
• Oxygen saturation: 96% on room air

Laboratory results:
• Serum glucose: 400 mg/dL (on arrival at the emergency department)
• Serum sodium (Na+): 125 mEq/L
• Serum potassium (K+): 6.5 mEq/L
• Serum chloride (Cl): 120 mEq/L
• Blood urea nitrogen (BUN): 39 mg/dl
• Serum creatinine (Cr): 1.7 mg/dl
• Urinalysis – positive for ketones

Arterial blood gas (ABG) results:
• pH: 7.30
• PaCO2: 33 mm Hg
• PaO2: 95 mm Hg
• HCO3: 19 mEq/L

The client is admitted to the intensive care unit, and you are the nurse assigned to the client. Reply to the following questions:
1. Based on an interpretation of the ABG results, what acid-base imbalance is occurring?
2. What nursing assessment findings are expected?
3. Describe strategies for safe, effective multidimensional nursing care of the client. Include rationale to support the strategies identified.

 

Sample Answer

Sample Answer

 

 

Nursing Care for a Client with Diabetic Ketoacidosis

1. Acid-Base Imbalance Interpretation

Based on the provided arterial blood gas (ABG) results, the client is experiencing metabolic acidosis. The key findings supporting this conclusion include:

– pH: 7.30 (normal range: 7.35 – 7.45) indicates acidosis.
– HCO3: 19 mEq/L (normal range: 22 – 26 mEq/L) shows a decreased bicarbonate level, which is characteristic of metabolic acidosis.
– PaCO2: 33 mm Hg (normal range: 35 – 45 mm Hg) suggests respiratory compensation, as the body is attempting to eliminate CO2 to counteract the acidosis.

2. Expected Nursing Assessment Findings

Given the client’s presentation and laboratory results, the following nursing assessment findings are expected:

– Vital Signs:

– Tachycardia (Pulse: 110 beats/minute) and hypotension (BP: 90/60 mm Hg) indicating possible shock or dehydration.
– Tachypnea (Respirations: 32 breaths/minute) as a compensatory mechanism for metabolic acidosis (Kussmaul respirations may also be observed).

– Neurological Status:

– Lethargy or altered level of consciousness due to hyperglycemia and metabolic derangements.

– Abdominal Assessment:

– Abdominal pain may indicate diabetic ketoacidosis (DKA) or gastrointestinal distress.

– Skin Assessment:

– Dry mucous membranes and poor skin turgor due to dehydration from osmotic diuresis.

– Laboratory Results:

– Hyperglycemia (serum glucose: 400 mg/dL).
– Electrolyte imbalances, particularly hyperkalemia (Serum potassium: 6.5 mEq/L) and hyponatremia (Serum sodium: 125 mEq/L).
– Elevated BUN and creatinine, indicating potential acute kidney injury secondary to dehydration.

3. Strategies for Safe, Effective Multidimensional Nursing Care

a. Continuous Monitoring

Strategy: Implement continuous monitoring of vital signs, blood glucose levels, electrolytes, and neurological status.

Rationale: Continuous assessment allows for early identification of deterioration or improvement in the client’s condition and facilitates timely interventions. Monitoring trends in vital signs can provide insights into the effectiveness of treatment interventions.

b. Administer Insulin Therapy

Strategy: Administer intravenous (IV) insulin as prescribed to lower serum glucose levels gradually.

Rationale: Insulin therapy will help shift glucose into cells and decrease ketogenesis, correcting hyperglycemia and metabolic acidosis. It also helps regulate potassium levels by driving potassium back into cells.

c. Fluid Resuscitation

Strategy: Initiate IV fluid therapy with isotonic saline to rehydrate the client, followed by a switch to hypotonic solutions as needed.

Rationale: Fluid resuscitation is crucial in DKA to restore intravascular volume, improve renal perfusion, and dilute serum glucose and electrolyte concentrations. It also addresses the dehydration resulting from osmotic diuresis.

d. Electrolyte Management

Strategy: Monitor serum potassium levels closely and administer potassium supplements as indicated.

Rationale: Hyperkalemia is a concern in DKA due to acidosis and insulin deficiency; however, potassium levels may drop rapidly with insulin administration, so it is vital to adjust potassium supplementation accordingly to prevent hypokalemia.

e. Education and Counseling

Strategy: Provide education about diabetes management, including the importance of adhering to insulin therapy and dietary recommendations.

Rationale: Empowering the client with knowledge about their condition can promote adherence to treatment plans, reduce the risk of future complications, and improve overall health outcomes.

f. Interdisciplinary Collaboration

Strategy: Collaborate with the healthcare team, including physicians, dietitians, and diabetes educators.

Rationale: An interdisciplinary approach ensures comprehensive care addressing all aspects of the client’s condition, including medical management, nutritional support, and education for long-term diabetes management.

Conclusion

The management of a client with diabetic ketoacidosis requires vigilant nursing care focused on monitoring, therapeutic interventions, education, and collaboration within a multidisciplinary team. By employing these strategies, nurses can significantly impact the client’s recovery and long-term diabetes management success.

 

 

 

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