Cardiovascular
Mr. W.G. is a 53-year-old white man who began to experience chest discomfort while playing tennis with a friend. At first, he attributed his discomfort to the heat and having had a large breakfast. Gradually, however, discomfort intensified to a crushing sensation in the sternal area and the pain seemed to spread upward into his neck and lower jaw. The nature of the pain did not seem to change with deep breathing. When Mr. G. complained of feeling nauseated and began rubbing his chest, his tennis partner was concerned that his friend was having a heart attack and called 911 on his cell phone. The patient was transported to the ED of the nearest hospital and arrived within 30 minutes of the onset of chest pain. In route to the hospital, the patient was placed on nasal cannula and an IV D5W was started. Mr. G. received aspirin (325 mg po) and 2 mg/IV morphine. He is allergic to meperidine (rash). His pain has eased slightly in the last 15 minutes but is still significant; was 9/10 in severity; now7/10. In the ED, chest pain was not relieved by 3 SL NTG tablets. He denies chills.

Case Study Questions

For patients at risk of developing coronary artery disease and patients diagnosed with acute myocardial infarct, describe the modifiable and non-modifiable risk factors.
What would you expect to see on Mr. W.G. EKG and which findings described on the case are compatible with the acute coronary event?
Having only the opportunity to choose one laboratory test to confirm the acute myocardial infarct, which would be the most specific laboratory test you would choose and why?
How do you explain that Mr. W.G temperature has increased after his Myocardial Infarct, when that can be observed and for how long? Base your answer on the pathophysiology of the event.
Explain to Mr. W.G. why he was experiencing pain during his Myocardial Infarct. Elaborate and support your answer.

 

Sample Answer

Sample Answer

 

Cardiovascular Health: A Critical Case Study Analysis

Introduction

Cardiovascular diseases, including coronary artery disease and acute myocardial infarction, are among the leading causes of morbidity and mortality worldwide. In this case study, we explore the medical emergency experienced by Mr. W.G., a 53-year-old man presenting with symptoms suggestive of an acute coronary event. Through an in-depth analysis, we aim to elucidate the risk factors, diagnostic approaches, and pathophysiological mechanisms underlying his condition.

Risk Factors for Coronary Artery Disease and Acute Myocardial Infarction

Modifiable Risk Factors:

– Hypertension: Uncontrolled high blood pressure can strain the heart and arteries.
– Hyperlipidemia: Elevated cholesterol levels contribute to atherosclerosis.
– Diabetes: Poorly managed diabetes increases the risk of cardiovascular complications.
– Smoking: Tobacco use accelerates atherosclerotic plaque formation.

Non-Modifiable Risk Factors:

– Age: Increasing age is a significant risk factor for cardiovascular diseases.
– Gender: Men are generally at higher risk for coronary artery disease at a younger age.
– Family History: Genetic predisposition plays a role in cardiovascular risk.

EKG Findings and Acute Coronary Event

On Mr. W.G.’s EKG, one would expect to see:

– ST-Segment Elevation: Indicates acute myocardial infarction.
– T-Wave Inversions: Can suggest ischemia or infarction.
– Q-Waves: Signify prior myocardial damage.

The crushing chest pain spreading to the neck and jaw, unrelieved by nitroglycerin, is compatible with an acute coronary event.

Specific Laboratory Test for Acute Myocardial Infarction

The most specific laboratory test to confirm acute myocardial infarction is Troponin. Troponin levels rise within hours of myocardial injury and remain elevated for an extended period, providing a reliable indicator of cardiac damage.

Temperature Changes after Myocardial Infarction

After a myocardial infarction, Mr. W.G.’s increased temperature can be attributed to the inflammatory response triggered by cardiac tissue damage. This fever may be observed for a few days post-infarction as part of the healing process.

Explanation of Pain during Myocardial Infarction

During a myocardial infarction, the intense chest pain experienced by Mr. W.G. is due to ischemia (lack of blood flow) and subsequent necrosis of cardiac muscle tissue. The release of inflammatory mediators and stimulation of pain receptors in the heart’s lining contribute to the severe, crushing pain characteristic of a heart attack.

In conclusion, the case study of Mr. W.G. underscores the critical nature of prompt recognition and management of acute coronary events. By understanding the multifaceted aspects of cardiovascular health, healthcare providers can effectively intervene to mitigate risks, diagnose accurately, and provide optimal care for patients experiencing cardiac emergencies.

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